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By C. Huang

ISBN-10: 9533079266

ISBN-13: 9789533079264

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2002). The circulating levels of ACE are unchanged in obesity as is its gene expression in adipose tissue of humans but there is a positive correlation with blood pressure (Gorzelniak et al, 2002). It has been difficult to get evidence for a key role of ACE but recently it was reported that ACE inhibition using captopril treatment of mice on a high fat diet reduced the extent of obesity and the expression of markers of inflammation in murine adipose tissue (Premaratna et al. 2011). Lee at al (2008a) reported that in obese rats, angiotensin receptor blockade reduced insulin resistance by modification of adipose tissue metabolism.

28 Secretory type II phospholipase A2 [PLA2] PLA2 is an acute phase protein that is able to degrade phospholipids present in lipoproteins and cell membranes thus releasing inflammatory molecules (Rosenson and Gelb, 2009). , 1999). , 2002; Fain, 2011). , (2011) failed to see elevations in circulating PLA2 in humans with severe coronary artery disease or any difference in its gene expression in epicardial, sternal or substernal fat of controls as compared to those with coronary artery disease. I conclude that PLA2 is primarily an obesity marker.

2011) reported decreases in obesity. , (2010) concluded that there is no consensus on the relationship between BMI or other cardiovascular risk factors and the circulating levels of OPG much less the function of this protein in humans. 27 Plasminogen activator inhibitor protein-1 [PAI-1] PAI-1 is also known as serpin E1 and is a member of the serpin family of serine protease inhibitors. , 2007). , 2004a). Total release by human fat in vitro correlates with BMI (Fain, 2010) and the gene expression of PAI-1 in visceral omental fat of obese women correlated to a greater extent with BMI than that of inflammatory adipokines such as TNF, MCP-1, IL-1, IL-6, IL-8 or MIF (Fain, 2011).

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Advanced Bariatric and Metabolic Surgery by C. Huang

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